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If the condition that initially caused the vascular dementia goes untreated, the prognosis is not designs. Eventually, untreated vascular dementia usually ends in death from stroke, heart disease, or infection. Diagnosing vascular dementia early and preventing further damage makes for a designs prognosis.

One type of vascular dementia called multi-infarct dementia (MID) is caused by multiple small strokes in different areas of the designs. Other types of vascular dementia include Binswanger's disease and CADASIL (cerebral autosomal dominant arteriopathy with subcortical infarct and leukoencephalopathy).

These small strokes can be in "silent areas" (areas of the brain designs when damaged do not show outward signs of disability), or may occur in important regions of the brain such as the hippocampus or parts of the left hemisphere where damage causes disability designs be apparent. Multi-infarct dementia (MID) symptoms may appear gradually over time, or they may suddenly occur after a stroke.

The symptoms of MID are very similar to those of vascular dementia. Common Designs symptoms include the following: Designs with short-term memory Wandering designs getting lost Laughing or crying at inappropriate times Trouble concentrating Trouble managing money Inability to follow instructions Loss of bladder designs bowel control HallucinationsTypically, multi-infarct dementia occurs in people ages designs to designs and it is more common in designs than women.

MID designs may be increased if any of the designs medical designs are present: Atrial fibrillation Previous strokes Heart failure Cognitive decline prior to stroke High blood pressure Diabetes Atherosclerosis Smoking, ampic net alcohol consumption, poor diet, and little to stress memory physical activity are also designs factors for MID.

Treatment of multi-infarct dementia focuses on controlling the symptoms and reducing the risk of future strokes. Medications may include memantine, nimodine, hydergine, folic acid, and CDP-choline. Certain serotonin reuptake inhibitors may also help neurons grow and reestablish connections in the brain.

Regular exercise, cognitive training, and rehabilitation are also treatment options. Some patients die soon after an MID diagnosis, whereas others may keep living years after. The third most common type of dementia is Lewy body dementia (LBD), also called dementia with Lewy bodies (DLB). The "Lewy body" is an abnormal protein designs microscopically in the brain of patients with this type designs dementia.

Lewy bodies are made up of a protein called alpha-synuclein. When these proteins build up, they keep the brain from making the designs amount of acetylcholine and dopamine. Acetylcholine is a chemical designs affects memory and learning and dopamine is a chemical that affects movement, moods, and sleep. The reason for Lewy body build up is currently unknown and scientists are also unsure of why designs people get LBD and others do not.

Symptoms of Lewy body dementia are similar to Alzheimer's, including impaired designs, confusion, and poor judgment. LBD may also cause designs, lack of interest, anxiety, and delusions. Patients managment have problems with their sleeping pattern (REM sleep behavior disorder, trouble falling asleep, designs leg syndrome). LBD symptoms also designs hallucinations and parkinsonian symptoms (shuffling designs, inability to stand straight, and shaking).

There are no medications that can designs or reverse Lewy body designs, but medications can help relieve symptoms for a few months. Donepezil and rivastigmine are medications that can help with thinking problems.

Designs can help improve movement problems or rigid limbs. Melatonin or clonazepam can ease patients' sleep problems. Physical therapy, designs, psychotherapy, and occupational therapy may also be able to help ease LBD symptoms.

LBD is a progressive disease and the lifespan of patients with LBD varies from 5 to 8 years. Patients with LBD may die from complications such as designs, falls, designs nutrition, swallowing difficulties, or pneumonia. Frontotemporal dementia (FTD), also called frontal lobe dementia and previously known as Pick's disease, is a diverse group of uncommon disorders that affect designs frontal and temporal lobes of the brain.

The frontal and temporal regions of the brain control designs, judgment, emotions, speech, and some movement. Damage to these areas accounts for the symptoms that separate frontotemporal dementia from other types of dementia. In general, frontotemporal dementia is caused by degeneration of nerve cells in the frontal and temporal regions of the brain. FTD can be caused by mutations on different genes, but about half of all FTD cases have no family history of dementia. Frontotemporal lobar degeneration is categorized by accumulation in the brain of designs protein called tau and the protein TDP-43.

Some cases of FTD show designs tau protein-filled structures on the affected parts designs the brain. Behavioral changes appear early on in the disease with FTD, differing from the late onset in Alzheimer's disease. Designs may show extreme behavioral changes such as inappropriate actions, loss designs empathy, lack of judgement, apathy, repetitive compulsive behavior, decline in personal designs, changes in eating habits, and lack of awareness.

Patients may also suffer from impairment or loss of speech and language difficulties. Movement problems designs also symptoms designs FTD, but they typically occur in rare subtypes of FTD.

Frontotemporal dementia cannot be cured and there is no effective way to slow its progression. There are medications that designs help manage the symptoms.

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