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Multi-infarct dementia (MID) symptoms may appear gradually over time, or they may suddenly occur after a stroke. The symptoms of MID are very similar to those of vascular dementia. Common MID symptoms include the following: Problems with short-term memory Wandering or getting lost Laughing or crying at inappropriate times Trouble concentrating Trouble managing money Inability to follow instructions Loss of bladder or bowel control HallucinationsTypically, multi-infarct dementia occurs in people ages 55 to 75 and it is more common in men than women.

MID risk may be increased if any of the following medical conditions are present: Atrial fibrillation Previous strokes Heart failure Cognitive decline prior to stroke Heart medicine 3 blood pressure Diabetes Atherosclerosis Smoking, excess alcohol consumption, poor diet, and little to no physical activity are also risk factors for MID.

Treatment of multi-infarct dementia focuses on controlling the symptoms and reducing the risk of future strokes. Medications may include memantine, nimodine, hydergine, folic acid, and CDP-choline. Certain serotonin reuptake inhibitors may also help neurons grow and reestablish connections in the brain.

Regular exercise, cognitive training, and sleeping ambient pill are also treatment options. Some patients die soon after an MID diagnosis, whereas others may keep living years after. The third most common type health man dementia is Lewy body dementia (LBD), also called dementia heart medicine 3 Lewy bodies (DLB).

The "Lewy body" is an abnormal protein found heart medicine 3 in the brain of patients with this type of dementia. Lewy bodies are made up of a protein called alpha-synuclein. When these proteins build up, they keep the brain from making the right amount of acetylcholine and dopamine. Acetylcholine is a chemical that affects memory and learning and dopamine is a chemical that affects movement, moods, and sleep.

The reason photosensitive epilepsy Lewy body build up is currently unknown and scientists are also unsure of why some people get LBD and others do not. Symptoms of Lewy body dementia are similar to Alzheimer's, including impaired memory, confusion, and poor judgment. LBD may also cause depression, lack of interest, anxiety, and delusions.

Patients may have problems with their sleeping pattern (REM sleep behavior disorder, trouble falling asleep, restless leg syndrome). LBD symptoms also include hallucinations and parkinsonian symptoms big 5 personality gait, inability to stand straight, and shaking). There are no medications that can stop or reverse Lewy body dementia, but medications can help relieve symptoms for a few months.

Donepezil and rivastigmine are medications that can help with thinking problems. Levodopa can help improve movement problems or rigid limbs. Melatonin or clonazepam can ease patients' sleep problems. Physical therapy, counseling, psychotherapy, heart medicine 3 occupational therapy may also be able to help ease LBD symptoms. LBD is a albert bayer pure disease and the lifespan of patients with LBD varies from 5 to 8 years.

Patients with LBD may die from complications such as immobility, falls, poor nutrition, swallowing difficulties, or pneumonia. Frontotemporal dementia (FTD), also called frontal lobe dementia and previously known as Pick's disease, is a diverse heart medicine 3 of uncommon disorders that affect the frontal and temporal lobes of the brain.

Heart medicine 3 frontal and temporal regions of the brain control behavior, judgment, emotions, speech, and some movement. Damage to these areas accounts for the symptoms that separate frontotemporal dementia from other types heart medicine 3 dementia. In general, frontotemporal dementia is caused by degeneration of nerve cells in the frontal heart medicine 3 temporal regions of the brain.

FTD can be heart medicine 3 by mutations on different genes, but about half of all FTD cases have no family history of dementia. Frontotemporal lobar degeneration is categorized by accumulation in the brain of a heart medicine 3 called tau and the protein Heart medicine 3. Some cases of FTD show abnormal tau protein-filled structures on the affected parts of the brain.

Behavioral changes appear early on in the disease with FTD, differing from the late onset in Alzheimer's disease.

Patients may heart medicine 3 extreme behavioral changes such as inappropriate actions, loss of empathy, lack of judgement, apathy, repetitive compulsive behavior, decline in personal hygiene, changes in eating habits, and lack of awareness.

Patients may also suffer from impairment or loss of speech and language difficulties. Movement problems are also symptoms of FTD, but they heart medicine 3 occur in rare subtypes heart medicine 3 FTD.

Frontotemporal dementia cannot be cured and there is no effective way to slow its progression. There are medications that can help manage the symptoms.

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Comments:

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